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MAURICE SCHIFF, MD; MARTIN MAGI, MD; ALLAN M. WARNER, MD

Arch Otolaryngol. 1963;77(6):595-597.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

For many years facial nerve decompression procedures have been concerned primarily with intratemporal lesions produced by chronic mastoiditis, middle-ear disease, mastoid surgery, and Bell's palsy, the lesions being located distal to the geniculate ganglion.

In our highly motorized society with the resultant increased incidence of severe head trauma, attention must also be focused on the intracranial facial nerve lesions, thus leading into an area with many unsolved problems.

The attack on intracranial facial nerve injuries was particularly stimulated by the work of Clerc and Batisse,¹ Dott,² and Kettel.³ However, it remained for House⁴ to reemphasize the detailed anatomical aspects and to devise an ingenious, technically feasible approach to the "gray zone" of facial nerve surgery.

In the facial nerve paralysis due to basilar skull fractures, the site of injury may involve the vertical segment of the nerve, the area near the bend, or the tympanic segment—all . . . [Full Text PDF of this Article]

Author Affiliations
OAKLAND, CALIF

From the Ear, Nose and Throat Service and Neurosurgery Branch of the Surgical Service, US Naval Hospital, Oakland. The opinions or assertions contained herein are the private ones of the writers and are not to be construed as official or as necessarily reflecting the views of the Medical Department of the Navy or of the Naval Service at large.

Captain, MC, USN (Ret.), present address: Connective Tissue Research Laboratory, University of Copenhagen, Copenhagen, Denmark (Dr. Schiff); Lieutenant, MC, USN, Resident in Otolaryngology, Ear, Nose and Throat Service, US Naval Hospital, Oakland (Dr. Magi); Lieutenant Commander, MC, USNR, Neurosurgery Branch, US Naval Hospital, Oakland (Dr. Warner).

Footnotes
Submitted for publication Nov 23, 1962.

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