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CÉSAR FERNÁNDEZ, M.D.; RENÉ ALZATE, M.D.

AMA Arch Otolaryngol. 1959;69(1):82-94.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Several workers have investigated the susceptibility of cochlear potentials to oxygen deprivation. The results showed that cochlear microphonics, action potentials, and DC resting potential are oxygen-dependent. Békésy¹ demonstrated that both cochlear microphonics and DC resting potential, as recorded from scala media, followed the same time-course decay during anoxia. Wever et al.² reported that in the cat the amplitude of cochlear microphonics diminished when the concentration of oxygen in the respirating gas mixture was below 4%. Lawrence and Wever³ reported a detectable lesion in the organ of Corti of cats exposed to severe oxygen deprivation. However, Falbe-Hansen et al.⁴ did not find such changes in guinea pigs and cats exposed to either acute or prolonged oxygen lack. Bornschein and Gernandt⁵ showed that by repeating the exposure to anoxia, the neural components can be removed selectively from cochlear microphonics. But according to other investigations⁶ this method . . . [Full Text PDF of this Article]

Author Affiliations
Chicago

From the Division of Otolaryngology of the University of Chicago.

Footnotes
Submitted for publication May 20, 1958.

Partially supported by Research Grant B-682 (C3) from the National Institute of Health and by Contract AF 41 (657) 143 with the School of Aviation Medicine, U. S. A. F., Randolph Field Air Force Base, Texas.

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