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GERHARD D. STRAUS, M.D.

Arch Otolaryngol. 1950;51(2):165-171.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

THE ROLE of abdominal infection in the production of benign esophageal stricture has long been a topic of discussion. Mosher stated the belief that fibrosis of the esophagus can develop from an infectious thrombophlebitis of the esophageal and periesophageal veins originating from infections of the liver and the gallbladder. Benign strictures of the esophagus associated with duodenal ulcer have been reported by various authors.

Larson, Layne and Howard¹ reported such a case. Their patient had a typical history of duodenal ulcer and roentgen evidence of ulcer. A posterior gastroenterostomy was performed. At the end of the fourth postoperative week dysphagia began to be apparent. Roentgen examination showed an almost complete obstruction in the lowest third of the esophagus, about 2 inches (5 cm.) above the diaphragm. Attempts were made to dilate the stricture. A massive pleural effusion containing large numbers of hemolytic streptococci suddenly developed and the patient died. Postmortem . . . [Full Text PDF of this Article]

Author Affiliations
MILWAUKEE

From the Departments of Otolaryngology, Marquette University School of Medicine and Veterans Hospital, Wood, Wis.

Footnotes
Presented as a candidate's thesis to the American Laryngological, Rhinological and Otological Society, Inc., April 1949.

Published with the permission of the Chief Medical Director, Department of Medicine and Surgery, Veterans Administration, who assumes no responsibility for the opinions expressed and the conclusions drawn by the author.

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