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A Review

E. A. SPIEGEL, M.D.

Arch Otolaryngol. 1946;44(1):61-72.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

THE second World War has revived the interest in seasickness and airsickness. The chief signs and symptoms of these syndromes are vegetative disturbances such as vasomotor reactions, pallor of the face, perspiration, nausea and vomiting (Byrne¹). Although it is recognized that seasickness is due chiefly to abnormal excitation of the labyrinth (Kreidl² and James³) and a similar assumption is rather probable for airsickness (Armstrong⁴), symptomatologic clinical studies alone are insufficient in order to decide whether the aforementioned vegetative symptoms are secondary effects of vertigo and the associated emotional corticodiencephalic reactions or whether they are the direct reflex effect of excitation of the nonacoustic inner ear. Experiments on animals are able to demonstrate the reflex nature of the majority of these vegetative reactions.

VASOMOTOR REACTIONS

The certain evidence that stimulation of the labyrinth leads to a vasomotor reaction is furnished by experiments in which the blood pressure in rabbits was recorded . . . [Full Text PDF of this Article]

Author Affiliations
PHILADELPHIA

Department of Experimental Neurology, Temple University School of Medicine.

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