Gruber,1 in 1890, spoke of the effect of increased intralabyrinthine pressure being transmitted through the saccus endolymphaticus to the cerebellum as a cause of vertigo.
In 1913, I2 called attention to the saccus endolymphaticus as a safety valve for increasing intralabyrinthine pressure.
Portmann,3 in 1921, experimenting with the ductus endolymphaticus in selaciens, which in them opens on the exterior, near and a little behind the eye, found that this fish after closing these openings by cauterization, acted, for a certain period of time, as though the static labyrinths had lost their functions.
At the meeting of the Eastern Section of this society, January 23, of this year, I explained labyrinthine tonus as the result of intracranial pressure waves transmitted directly to the vestibular nerve end organs through the ductus endolymphaticus.
The membranous labyrinth is a closed series of cavities which act as receptacles for the nerve terminations
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