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AN ANALYSIS OF FORTY-NINE CASES WITH A REPORT OF OBSERVATIONS AT NECROPSY IN ELEVEN

JOSEPH G. DRUSS, M.D.; BENJAMIN ALLEN, M.D.

Arch Otolaryngol. 1942;36(1):12-22.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The belief that there is impairment of immunity to infection in the tissues of the diabetic patient has been widely held for many years. In the field of otology this concept finds expression in the doctrine that in the presence of diabetes infections in the mastoid process are associated with relatively early and extensive bone destruction and are particularly prone to intracranial complications. Recently, however, exception has been taken to this view,¹ and it has been asserted that with the present improved therapy in diabetes, with especial reference to the use of insulin and a dietary regimen, the course of mastoiditis in the diabetic patient does not differ essentially from that in one who does not have diabetes. In an attempt to clarify this matter and to obtain, if possible, a better understanding of the influence of diabetes on the clinical manifestations, pathologic changes and treatment of mastoiditis, we . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Otologic Service and the Laboratories of the Mount Sinai Hospital.

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