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ASSOCIATED WITH PARALYSIS OF THE EXTERNAL RECTUS

I. FRIESNER, M.D.; J. G. DRUSS, M.D.

Arch Otolaryngol. 1930;12(3):342-365.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Whatever the cause of these differences, there are wide variations in the number, extent and size of the cells in the temporal bone. As a general rule, the larger the cells the greater is the facility with which infection spreads through them. In the highly pneumatized mastoid processes it is not an unusual operative observation to uncover purulent disease in cells remote from the antrum. It is not clear why infections in the mastoid vary in their predilection for certain pathways. Neumann,¹ however, called attention to this fact.

It is particularly true of the extensively pneumatized, large cell mastoids that the pneumatic structure extends about the bony labyrinthine capsule into the depths of the petrous pyramid. Just as the cells in the mastoid vary in number, extent and size, so do the cells of the petrosa; furthermore, just as in the mastoid an infection at times exercises . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Otological and Pathological Departments of Mount Sinai Hospital.

Footnotes
Submitted for publication, April 26, 1930.

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